Trans-FA inhibits the proliferation of H1299 lung cancer cells and induces a reasonable increase in the apoptotic populace by advertising phosphorylation of β-catenin at residues Thr41 and Ser45 and producing proteasomal degradation [117,177]. Intriguingly, FA is capable of inhibiting the proliferation and migration of lung cancer cells by eradicating intracellular https://confuciusu000jtf3.wikicorrespondent.com/user